Nurse Advocate: Case Study

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Showing posts with label Case Study. Show all posts
Showing posts with label Case Study. Show all posts

Thursday, July 28, 2011

Therapeutic Exercise


PhotoCredit: gbfamilycarechiropractic.com
Definition
Therapeutic exercises are activities that are prescribed by the physician and are performed with the assistance and guidance of a physical therapist or a nurse. The ultimate goal of therapeutic exercises is to help and aid the patient or injured individual gain independence in functional activities and make the activity as much as possible comfortable and pain-free.

Goals:

  • Enable ambulation
  • Release contracted muscles, tendons, and fascia
  • Mobilize joints
  • Improve circulation
  • Improve respiratory capacity
  • Improve coordination
  • Reduce rigidity
  • Improve balance
  • Promote relaxation
  • Improve muscle strength and, if possible, achieve and maintain maximal voluntary contractile force (MVC)
  • Improve exercise performance and functional capacity (endurance)

Physiological Aspects of Physically Fit Person

  • Higher oxygen consumption (mL/min)
  • Greater cardiac output per minute
  • Higher stroke volume and total blood volume
  • Greater oxygen extraction by the tissues
  • Greater cardiac volume
  • Lower resting pulse rate
  • Greater muscle strength
  • Lower pulse rate on exercise
  • Lower blood pressure on exercise
  • Better adaptation of circulation and respiration to effort

Benefits of Therapeutic Exercises
When correctly performed, therapeutic exercises will have the following outcomes:
  1. Improve or maintain muscle strength. It is only through the execution of these exercises that muscle strength is built and reinforced.
  2. Enhancing joint function.
  3. Helps in preventing deformities.
  4. Assists in stimulating blood circulation.
  5. Developing a person’s endurance.
  6. Promoting relaxation.
  7. Helping to restore motivation and well-being of the patient
Approach Considerations
Therapeutic exercises aimed at achieving and maintaining physical fitness fall into the following major categories, each of which has a specific purpose:

  • Endurance training
  • Resistance training
  • Flexibility training
Endurance Training
An endurance training program has 3 variables: frequency, intensity, and duration. The American College of Sports Medicine (ACSM) recommendations are as follows:

  • Frequency - Aerobic exercise 3-5 days a week
  • Intensity of training - 64/70-94% of maximum heart rate (HR max), or 40/50-85% of maximum oxygen uptake reserve (VO2 R) or heart rate reserve (HRR)
  • Duration of training - Continuous or intermittent aerobic activity for 20-60 minutes (minimum of 10-min bouts accumulated throughout the day). Duration is dependent on the intensity of the activity; thus, lower-intensity activity should be conducted over a longer period of time (≥30 min), and, conversely, individuals training at higher levels of intensity should train at least 20 minutes or longer. Moderate-intensity activity of longer duration is recommended for adults not training for athletic competition, because total fitness is more readily attained with exercise sessions of longer duration and because potential hazards and adherence problems are associated with high-intensity activity.
Application to healthy individuals
Exercises that use large muscle groups that can be maintained continuously and are aerobic in nature are recommended. These exercises include walking, running, jogging, dancing, stair climbing, cycling, swimming, rowing, skating, aerobic dance/exercise classes, jumping rope, and cross-country skiing.

The HR max can be determined by the following: HR max = 220 – Ag

The exercise session should consist of the following:
  • A warm-up period of approximately 10 minutes: This should combine calisthenic-type stretching exercises and progressive aerobic activity that should increase the heart rate close to the prescribed heart rate for the session.
  • Endurance training (20-60 min)
  • A cool-down period of 5-10 minutes
Application to patients
For at-risk patients, especially those with cardiac or respiratory disease, a less intense training regimen must be used, with the training heart rate not exceeding that attained at 50-60% of maximum O2 uptake (VO2 max).

In the absence of data regarding maximum attained heart rate measurements, it is prudent not to allow a patient to exceed a heart rate of 130 beats per minute (bpm). In elderly patients and patients at risk, the intensity, frequency, and duration of therapeutic exercise should be established for each patient individually through prior medical evaluation (see Medical evaluation).
Using the following equation, the HRR method, otherwise known as the Karvonen method, should be employed to determine the target heart rate for the ill or elderly patient: Target HR = (220 - age - resting heart rate × % intensity selected) + resting heart rate

Progression
Progression must be a part of an exercise program to ensure continued results. With endurance training, progression can occur by increasing the duration or the intensity. Several factors contribute to the optimal rate of progression; current activity levels, exercise goals, age, and physiologic limitations should be considered. Most importantly, a rate of progression should be used that results in long-term participation. Being too aggressive with progression can lead to increased dropout rates as a result of injuries and/or perceived excessive discomfort.


Resistance Training
Resistance training increases strength, walking speed, stair-climbing power, balance, and lean body mass and decreases regional and total fat mass. This form of exercise has been shown to bring about favorable changes in risk factors for coronary artery disease, osteoporosis, diabetes mellitus, and cancer. For example, resistance training has been proven to lower systolic blood pressure, increase bone mineral density, increase mechanisms involved with blood glucose metabolism (glucose tolerance and insulin resistance), and increase bowel transit time, which decreases the risk of colon cancer in healthy men and women who are middle-aged or older. Back pain and work-related back injuries also have been shown to decrease with resistance training.


Factors Affecting the Adherence to Therapeutic Program
A therapeutic exercise program is beneficial to the patient. However, some individuals do not adhere to the program. The following are some of the identified factors that affect the performance of therapeutic exercise:
    • Lack of knowledge about the exercise program
    • Lack of Skill. Explaining and demonstrating the methods or techniques of exercise execution may be helpful in assuring adherence to the exercise program. Though the patient may indicate familiarity with the exercise, the technique execution might be wrong. An exercise is ineffective if done incorrectly. Thus, it is ideal to demonstrate the techniques and skill to the patient beforehand and let the patient perform the complete set of exercise while watching to correct improper technique execution. Correcting errors can be easily provided by positive reinforcement and suggestions.
    • Lack of Risk Taking Ability. An individual’s fear of feeling increased pain, discomfort or possible reinjury sometimes limits the individual’s motivation to exert full effort and thus it prevents the patient from sticking to the therapeutic exercise program. Emotions are one factor that may motivate or block the individual to deal successfully with the challenges of the exercise program. Nurses should provide constant positive feedback to deal with this.
    • Lack of Social support. When an individual feels alone or isolated, he or she might lose the motivation to meet the challenges of the exercise program. Assuring and acknowledging that value of a patient in an activity and encouraging socialization are the key steps in handling this problem.
    • Confidence. One of the most essential factors that affect the completion of the exercise program is the confidence of the person.  Timely recovery will be delayed when a person has low confidence because focus is interfered with negative thoughts.
    • Motivation. The greatest influence in the completion of the exercise program is based on how motivated an individual is. Low motivation results to the following:

    1. Low effort and intensity
    2. Poor attention
    3. Lack of attentiveness to instructions
    4. Undefined goals
    5. Insupportable excuses to avoid the exercise program

Tuesday, March 23, 2010

Case Study: Polyhydramnios

Polyhydramnios
  • Abnormally large amount of amniotic fluid in the uterus.
  • Normal range from 500 to 1,000 ml at term; typically greater than 2,000 ml in polyhydramnios at 40 weeks’ gestation.
Also called hydramnios

  • Possible complications: 

    1. prolapsed umbilical cord when membranes rupture
    2. increased incidence of malpresentations
    3. increased perinatal mortality from fetal malformations and premature deliveries
    4. increased incidence of postpartum maternal hemorrhage
Kinds/Degrees of Polyhydramnios
1.       Mild Polyhydramnios – when amniotic fluid pockets is between 8 to 11 cm in vertical dimensions. (85%)
2.       Moderate Polyhydramnios – when amniotic fluid pockets is between 12 to 15 cm in vertical dimensions. (17%)

Predisposing Factors

  • Multiple pregnancy
  • Fetal abnormalities – esophageal atresia, anencephaly, spina bifida
  • Diabetes Mellitus

Pathophysiology

  • Normally, amniotic volume is maintained by a balance of fetal fluid production (lung liquid and urine) and fluid resorption (fetal swallowing and flow across the membrane to the fetus or the maternal uterus).
  • Fetal urine is the primary source of amniotic fluid with output at term ranging from 400 to 1,200 ml/day.
  • Fetal swallowing is believed to be the major route of amniotic fluidresorption.
  • With polyhydramnios, fluid accumulates because of a problem with the fetus’s ability to swallow or absorb the fluid or as a result of over production of urine.
  • Fluid may have increased gradually (chronic type) by the third trimester or rapidly (acute type) between 20 and 24 week’s gestation.

Causes

  • Exact cause is unknown in about 35% of all cases.
  • May be associated with:

    1. diabetes mellitus (about 25%)
    2. erythroblastosis ( about 10%)
    3. multiple gestations (about 10%)
    4. anomalies of the central nervous system (such as neural tube defects)
    5. GI anomalies such as tracheoesophageal fistula that prevent ingestion of the amniotic fluid (about 20%).

Assessment Findings

  • Depend on the length of gestation, the amount of amniotic fluid, and whether the disorder is chronic or acute.
  • Mild signs and symptoms; maternal abdominal discomfort, slight dyspnea, and edema of feet and ankles.
  • Severe signs and symptoms; severe dyspnea, orthopnea, and significant edema of the vulva, legs, and abdomen.
  • Symptoms common to mild and severe cases: uterine enlargement greater than expected for the length of gestation, and difficulty in outlining the fetal parts and in detecting fetal heart sounds.

Test Results

  • Ultrasonography shows evidence of excess amniotic fluid as well as underlying conditions.
  • Amniotic fluid index is 20 cm or greater.

Treatment

  • High protein, low sodium diet
  • Mild sedation
  • Indomethacin therapy – a drug that decreases the fetal urine formation. The side effect of indomethacin, as with other prostaglandin synthase inhibitors, is the potential premature closure of the ductus arteriosus.
  • Amniotomy – The fluid is removed by a needle inserted through the cervix. The danger of this procedure is cord proplapse and abruptio placenta. to prevent these complications, amniotic fluid must be removed gradually.Watch closely for hemorrhage after delivery, prevent uterine relaxation by massaging the uterus and administering oxytoxin as ordered.
  • Induction of labor if the fetus is mature and symptoms are severe.

Nursing Interventions

  • Mild to moderate degrees usually does not require treatment.
  • Hospitalization if symptoms are severe dyspnea, abdominal pain and difficult ambulation.
  • Maintain bed rest with sedation to make the situation endurable.
  • Monitor the patient for signs and symptoms of premature labor.
  • Monitor maternal vital signs and fetal heart rate frequently; report changes immediately.
  • Prepare the patient for amniocentesis and possible labor induction, as appropriate; keep in mind that amniocentesis for fluid removals is only temporary and may need to be done repeatedly.

Case Study: Oligohydramnios

Oligohydramnios
§ Severe reduction of amniotic fluid volume (typically less than 500 ml at term); highly concentrated urine.
§ Possibility of prolonged, dysfunctional labor (usually beginning before term).
§ Fetal risk: renal anomalies, pulmonary hyperplasia, hypoxia, increased skeletal deformities, and wrinkled, leathery skin.


Causes
§ Exact cause is unknown.
§ Any condition that prevents the fetus from making urine or that blocks urine from going into the amniotic sac.
§ Contributing factors: uteroplacental insufficiency, premature rupture of membranes prior to labor onset, maternal hypertension, maternal diabetes, intrauterine growth restriction, postterm pregnancy, fetal renal genesis, polycystic kidneys, and urinary tract obstructions.

Assessment
§ Asymptomatic
§ Lagging fundal height growth.

Test result
§ Ultrasonography reveals no pockets of amniotic fluid larger than 1 cm.

Treatment
§ Close medical supervision of the mother and fetus.
§ Fetal monitoring
§ Amnioinfusion (infusion of warmed sterile normal saline or lactated Ringer’s solution) to treat or prevent variable decelerations during labor.

Nursing Interventions
1.     Monitor maternal and fetal status closely, including vital signs and fetal heart rate patterns.
2.     Monitor maternal weight gain pattern, notifying the health care provider if weight loss occurs.
3.     Provide emotional support before, during, and after ultrasonography.
4.     Inform the patient about coping measures if fetal anomalies are suspected.
5.     Instruct her about signs and symptoms of labor, including those she’ll need to report immediately.
6.     Reinforce the need for close supervision and follow up.
7.     Assist with amnioinfusion as indicated.
8.     Encourage the patient to lie on her left side.
9.     Ensure that amnioinfusion solution is warmed to body temperature.
10.  Continuously monitor maternal vital signs and fetal heart rate during the amnioinfusion procedure.
11.  Note the development of any uterine contractions, notify the health care provider, and continue to monitor closely.
12.   Maintain strict sterile technique during amnioinfusion.

Case Study: Blood Transfusion Therapy


Blood transfusion therapy involves transfusing whole blood or blood components (specific portion or fraction of blood lacking in patient). One unit of whole blood consists of 450 mL of blood collected into 60 to 70 mL of preservative or anticoagulant. Whole blood stored for more than 6 hours does not provide therapeutic platelet transfusion, nor does it contain therapeutic amounts of labile coagulation factors (factors V and VIII).


    Monday, January 18, 2010

    Case Study: What is Anxiety?

    Recognizing anxiety and the Levels of Anxiety with corresponding Nursing Interventions for each level. (www.nursingcrib.com)



    Case Study: Kubler-Ross Stages of Dying or Grief

    The Stages of Grief conceptualized by Kubler and Ross explained, with appropriate Nursing Interventions. (www.nursingcrib.com)


    Sunday, January 17, 2010

    Case Study: Pregnancy Complications

    An enumeration of pregnancy complications with corresponding causes and predisposing factors. Complications include: (www.nursingcrib.com)
    • Abortion
    • Ectopic Pregnancy
    • Hyperemesis Gravidarum
    • Placenta Previa
    • Abruptio Placenta
    • Pregnancy Induced Hypertension
    • Gestational Diabetes
    • Anemia
    • Hyatidiform Mole
    • Incompetent Cervix
    • Polyhydramnios
    • Oligohydramnios
    • Premature Labor


    Case Study: Pressure Sores

    An article featuring Pressure Sores, with its Assessment, Diagnostic Evaluation, including Therapeutic, Pharmacologic and Nursing Interventions. (www.nursingcrib.com)



    Case Study: Dialysis

    The article is a feature artile about Dialysis with its Indications, Goals, Types and their corresponding complications. (www.nursingcrib.com)



    Monday, July 27, 2009

    Case Study: Spontaneous Abortion

    A. Description

    1. Spontaneous abortion is the expulsion of the fetus and other products of conception from the uterus before the fetus is capable of living outside of the uterus.
    2. Types of spontaneous abortions

    a. threatened abortionThreatened abortion - is characterized by cramping and vaginal bleeding in early pregnancy with no cervical dilation. It may subside or an incomplete abortion may follow.
    b. Imminent or inevitable abortion – is characterized by bleeding, cramping and cervical dilation. Termination cannot be prevented.
    c. Incomplete abortion – is characterized by expulsion of only part of the products of conception (usually the fetus). Bleeding occurs with cervical dilation.
    d. Complete abortion – is characterized by complete expulsion of all products of conception.
    e. Missed abortion – is characterized by early fetal intrauterine death without expulsion of the products of conception. The cervix is closed, and the client may report dark brown vaginal discharge. Pregnancy test findings are negative.
    f. Recurrent (habitual) abortion – is spontaneous abortion of three or more consecutive pregnancies.
    B. Etiology – Spontaneous abortion may result from unidentified natural causes or from fetal, placental or maternal factors.
    1. Fetal Factors
    a. Defective embryologic development
    b. Faulty ovum implantation
    c. Rejection of the ovum by the endometrium
    d. Chromosomal abnormalities
    2. Placental Factors
    a. Premature separation of the normally implanted placenta
    b. Abnormal placental implantation
    c. Abnormal placental function
    3. Maternal Factors
    a. Infection
    b. Severe malnutrition
    c. Reproductive system abnormalities (eg, incompetent cervix)
    d. Endocrine problems (eg, thyroid dysfunction)
    e. Trauma
    f. Drug ingestion
    C. Pathophysiology – The fetal or placental defect or the maternal condition results in the disruption of blood flow, containing oxygen and nutrients, to the developing fetus. The fetus is compromised and subsequently expelled from the uterus.
    D. Assessment Findings

    1. Associated findings – The client and family may exhibit a grief reaction at the loss of pregnancy, including:
    a. Crying
    b. Depression
    c. Sustained or prolonged social isolation
    d. Withdrawal
    2. Clinical Manifestations – include common signs and symptoms of spontaneous abortion.
    a. Vaginal bleeding in the first 20 weeks of pregnancy
    b. Complaints of cramping in the lower abdomen
    c. Fever, malaise or other symptoms of infection
    3. Laboratory and diagnostic study findings
    a. Serum beta hCG levels are quantitatively low
    b. Ultrasound reveals the absence of a viable fetus.
    E. Implementation

    1. Provide appropriate management and prevent complications
    a. Assess and record vital signs, bleeding and cramping of pain.
    b. Measure and record intravenous fluids and laboratory test results. In instances of heavy vaginal bleeding; prepare for surgical intevention (D & C) if indicated.
    c. Prepare for PhoGAM administration to an Rh-negative mother, as prescribed. Whenever the placenta is dislodged (birth, D & C, abruptio) some of the fetal blood may enter maternal circulation. If the woman is Rh negative, enough Rh-positive blood cells may enter her circulation to cause isoimminization, the production of antibodies against Rh-positive blood, thus endangering the well-being of future pregnancies. Because the blood type of the conceptus is not known, all women with Rh-negative blood should receive RhoGAM after an abortion.
    d. Recommended iron supplements and increased dietary iron as indicated to help prevent anemia.
    2. Provide client and family teaching
    a. Offer anticipatory guidance relative to expected recovery, the need for rest and delay of another pregnancy until the client fully recovers.
    b. Suggest avoiding intercourse until after the next menses or using condoms when engaging in intercourse.
    c. Explain that in many cases, no cause for the spontaneous abortion is ever identified.
    3. Address emotional and psychosocial needs.

    Case Study: G6PD Deficiency

    What is G6PD deficiency?
    G6PDGlucose-6-phosphate dehydrogenase deficiency, or G6PD deficiency for short, is the most common “inborn metabolic disorder” in the world. This means that from the time a baby is born, there is already something wrong with how his body makes and breaks important substances. According to statistics, about 400 million people have G6PD deficiency, and it is most common in Africa, Southeast Asia and the Middle East.
    Babies with G6PD deficiency have very little or no enzyme called Glucose-6-Phosphate Dehydrogenase (G6PD). An enzyme is a kind of protein that speeds up chemical reactions in the body. The enzyme G6PD is especially important to red blood cells. If this enzyme is lacking or missing, red blood cells are easily destroyed.

    Another name for G6PD deficiency is favism because some people who have it, usually those living in the Meditteranean region, react very badly to fava beans

    What causes G6PD deficiency?
    In order to understand what causes G6PD deficiency, one must first learn a bit about genes and chromosomes.

    Genes are like the body’s blueprints. They contain instructions on how specific parts of the body are made. For example, if the instructions in your hair genes say your hair is black, your hair will be black. Genes are packaged into threadlike structures called chromosomes. A chromosome is very much like a beaded bracelet. The beads are the different genes that give instructions for different part of the body; the entire bracelet is the chromosome. Genes usually come and act in pairs. One member of a specific pair comes from the father, and the other member comes from the mother. The members of a pair are located on paired chromosomes.

    All normal human beings have 23 pairs of chromosomes. Each of the first 22 pairs contain the same number and kind of genes. The last and 23rd pair is the sex chromosomes. They are different from the first 22 pairs in that they do not have the same number and kind of genes. The sex chromosomes contain the genes that determine whether a baby will be a girl or a boy.

    There are 2 kinds of sex chromosomes, X and Y. All baby girls have two X chromosomes. All baby boys have one X and one Y. The gene that gives instructions on how G6PD is made is found in the X chromosome only, thus G6PD deficiency is described as X-linked.
    If a baby girl gets one defective G6PD gene from either of her parents, she will not have G6PD deficiency because she has another G6PD gene that can do the work (remember: a baby girl has two X chromosomes, thus two G6PD genes). But if she gets two defective G6PD genes from both her parents, she will have G6PD deficiency. On the other hand, a baby boy whose G6PD gene is defective will surely get G6PD deficiency because the Y chromosome has no G6PD gene.

    A defective G6PD gene will give wrong instructions on how to make the enzyme G6PD. As a result, too little or none of it is made.

    What are the harmful effects of G6PD deficiency?
    G6PD has a very small but strategic role in protecting the body from substances that can cause damage to cells or oxidative substances. Because of this important role, G6PD is normally found in all parts of the body. To be sure, most parts of the body also keep a “spare” enzyme, one that can do the work of G6PD in case it is lacking or missing entirely. Unfortunately, this is not the case with red blood cells. They do not have spare enzymes that can do the work of G6PD. If a baby does not have enough G6PD, his red blood cells lack protection from the harmful effects of oxidative substances.

    A baby with G6PD deficiency appears and remains healthy until he is exposed to a large amount of oxidative substances. When this happens, his red blood cells are destroyed, a process known as hemolysis.

    Red blood cells carry oxygen to all parts of the body. When they undergo hemolysis, the baby will have hemolytic anemia. The signs and symptoms of hemolytic anemia are paleness, dizziness, headache, tea-colored urine, and abdominal or back pain or both. Hemolytic anemia, when very severe, can end in death. Destroyed red blood cells are brought to the liver to be broken down to smaller pieces for disposal. One of the end products of this process is bilirubin, a yellowish substance that accumulates in different parts of the body when too much of it is produced. Quite often, bilirubin accumulates in the skin and causes it to appear yellowish. In the worst cases, biliribin accumulates in the brain and causes mental retardation or death.

    Where do oxidative substances come from?
    Hemolysis of red blood cells will only occur IF and WHEN a G6PD deficient child is exposed to oxidative substances. Oxidative substances are found in certain drugs, foods, and beverages. The body also produces oxidative substances during severe infections or illnesses such as typhoid fever, pneumonia, or kidney failure.
    Most drugs with strong oxidative effects are of kinds:
    1. antibiotics of the sulfa group
    2. medicines for malaria
    3. some medicines for fever

    How is G6PD deficiency treated?
    When a child has taken oxidative substances and suddenly shows the signs and symptoms of hemolytic anemia, he is said to have a hemolytic crisis. During such crisis, the goal of doctors and nurses is to prevent the harmful effects from getting worse. Blood transfusion, oxygen, and folic acid may be given.
    The ultimate treatment for G6PD deficiency is gene therapy (replacing a defective gene with a good one), but this is not yet available at the present time.

    As parent, what should I do to prevent a hemolytic crisis?
    1. Tell your child’s pediatrician that your child has G6PD deficiency. This is very important so that he will not prescribe oxidative drugs in case your child gets ill. He would also be able to watch out for hemolytic crisis and would immediately know what to do just in case it happens.
    2. Keep your list of oxidative substances in a handy place. Better yet, post it in a convenient spot on the kitchen wall. Always double-check food, beverage, and medicine labels against the list.
    3. Memorize the signs and symptoms of hemolytic anemia: paleness, dizziness, headache, difficulty in breathing, rapid and strong heartbeats, tea-colored urine, and abdominal or back pain. Bring your child to his pediatrician as soon as these signs and symptoms appear.
    4. Do not ignore infections. Persistent fever signals an infection. Bring the child at once to his pediatrician.
    5. As your child gets older, honestly and gently tell him about his condition and teach him to be careful about what he eats.

    IMPORTANT REMINDERS for G6PD deficiency Individuals
    1. If you have coughs, cold or other bacterial or viral infections, make sure to inform your doctor that your have G6PD.
    2. If you have ingested or were exposed to any medication and your urine became tea-colred inform your doctor immediately.
    3. If you have yellowish discoloration of your skin, sclera or any part of your body, consult your doctor immediately.
    4. Avoid the following foods that are contraindicated among G6PD deficient individuals:
    • fava beans
    • red wine
    • legumes (bitsuelas, garbansos, monggo beans)
    • blueberries (also applies to food products containing these)
    • soya foods (taho, tokwa, soy sauce)
    • tonic water
    Note: Except for fava beans, there is no adequate proof as yet of the hemolytic effects of these foods.
    5. Avoid ingestion of or exposure to the following drugs and chemicals:
    Generic Name
    Brand Names
    Acetanilid


    Chloroquine
    Aralen
    Maralex
    UL Chloroquine
    Doxorubicin HCl

    Adriblastina RD
    Adrim
    Biomedis
    Caelyx
    Doxorubicin HCI
    Doxorubicin Meiji
    Faulding/DBL
    K.U. Doxorubicin HCI
    Pfizer Doxorubicin
    Pharmachemie
    Pharmacia
    Doxorubicin HCI
    Rubidox
    Furazolidone
    Diafuran
    Diapectolin
    Drugmaker’s Biotech
    Furoxone
    Pseudoambin
    Methylene blue

    Menthol

    Alaxan Gel
    Begesic
    Ben-gay
    Broncho Rub White
    Efficascent Oil
    Listerine mouthwash
    Listerine Pocketpacks
    Mediplastin
    Megascent Oil
    Mentopas Medicated Plaster
    Metsal
    Omega Pain Killer
    Perskindol
    Rowachol
    Sarna
    Nalidixic Acid
    Hanadex
    Wintomylon
    Naphthalene


    Niridazole


    Nitrofurantoin
    Harfurin
    Macrodantin
    USA Lab Nitrofurantoin
    Transpulmin Balsam

    Phenazopyridine
    Azomir
    Phenylhydrazine


    Primaquine


    Quinidine
    Kinidin
    Sulfacetamide
    Acetopt
    Bleph 10
    Cetapred
    Isopto Cetamide
    Isopto Cetapred
    Sensocet
    Spersacet C
    Sultrin
    Sulfamethoxazole
    Bacidal
    Bactille Forte
    Bactille-TS
    Bactrim
    Bacxal
    Baczole
    Colimox
    Cotrexel
    Cotribase
    Cotrimoxazole
    Cotrimoxazole-Vamsler
    DLI Cotrimoxazole
    Doctrimox
    Drugmaker’s Biotech
    Fedimed
    Globaxol
    Gutrisol
    Intrafort
    Kathrex
    Lagatrim Forte
    Lictora
    Macromed
    Microbid/Microbid DS
    Moxadden
    Neotrim
    Onetrim
    Pharex Cotrimoxazole
    Procor
    Ritemed Cotrimoxazole
    Septrin
    Synerzole
    Thoprim
    Trihexal
    Trimephar
    Trimoxol
    Trim S
    Trizole suspension
    Xanozole
    Sulfanilamide


    Sulfapyridine


    Thiazolesulfone


    Toluidine Blue


    Trinitrotoluene


    Vitamin K
    Vitamin K with Adenogen
    Cycomin
    Hema-K
    Konakion MM/
    Konakion MM
    Paed

    Reference:
    1. Philippine Pharmaceutical Directory Review 5th Edition. Jocelyn J. Yambao et al. Ed. Medicom Pacific Inc. 2005.
    2. MIMS Philippines. Volume 31, Number 3, 2002. Medi Media. 2002.
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